科研之友
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摘要
In rodents, insufficient adenosine produces behavioral and physiological symptoms consistent with several comorbidities of autism. In rodents and humans, stimuli postulated to increase adenosine can ameliorate these comorbidities. Because adenosine is a broad homeostatic regulator of cell function and nervous system activity, increasing adenosine%26apos;s influence might be a new therapeutic target for autism with multiple beneficial effects. %26lt;br%26gt;This article is part of the Special Issue entitled %26apos;Neurodevelopmental Disorders%26apos;.
- 出版日期2013-5
