科研之友
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摘要
Evidence implicating the beta -amyloid protein (A beta) in the pathogenesis of Alzheimer's disease has steadily accumulated. However, the mechanism by which A beta causes dementia is unknown, Here we argue that a more integrated, top-down approach to brain function is needed to assess the role of A beta in Alzheimer's disease, and that more attention should be paid to the effects of A beta on synaptic function rather than on cell death.
- 出版日期2001-8
