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APOBEC3 Cytidine Deaminases in Double-Strand DNA Break Repair and Cancer Promotion
APOBEC3B Upregulation and Genomic Mutation Patterns in Serous Ovarian Carcinoma
APOBEC-Mediated Cytosine Deamination Links PIK3CA Helical Domain Mutations to Human Papillomavirus-Driven Tumor Development
APOBEC3 deletion polymorphism is associated with breast cancer risk among women of European ancestry
APOBEC3G exerts tumor suppressive effects in human hepatocellular carcinoma
APOBEC3D and APOBEC3F Potently Promote HIV-1 Diversification and Evolution in Humanized Mouse Model
APOBEC3 Deaminases Induce Hypermutation in Human Papillomavirus 16 DNA upon Beta Interferon Stimulation
APOBEC3G Inhibits HIV-1 RNA Elongation by Inactivating the Viral Trans-Activation Response Element
APOBEC3 signature mutations in chronic lymphocytic leukemia
APOBEC3G真核表达载体构建及抗HBV复制的初步研究
APOBEC3G在慢性乙型肝炎、肝癌组织中的表达及与HBsAg、HBcAg的关系
APOBEC3s对HPV(+)上皮细胞基因组稳定性的影响
APOBECs家族成员在癌症进化发育过程中的核心作用
APOB基因3′端短串联重复序列多态性与新疆维吾尔族自然长寿的关系
APOBEC家族:介导天然抗病毒免疫的新型宿主细胞因子
APOBEC-3F和APOBEC-3G与乙肝核心抗原的相互作用研究
APOBEC3A通过脱氨基作用抑制HBV复制
APOBEC3A and 3C decrease human papillomavirus 16 pseudovirion infectivity
APOBEC3B expression in drug resistant MCF-7 breast cancer cell lines
APOBEC3B is an enzymatic source of molecular alterations in esophageal squamous cell carcinoma
APOBEC-induced mutations in human cancers are strongly enriched on the lagging DNA strand during replication
APOBEC3A and APOBEC3B Preferentially Deaminate the Lagging Strand Template during DNA Replication
APOBEC3G Variants and Protection against HIV-1 Infection in Burkina Faso
APOBEC family mutational signatures are associated with poor prognosis translocations in multiple myeloma
APOBEC3B: A Potential Factor Suppressing Growth of Human Hepatocellular Carcinoma Cells
APOBEC3A Is Implicated in alpha Novel Class of G-to-A mRNA Editing in WT1 Transcripts
APOBEC3A Functions as a Restriction Factor of Human Papillomavirus
APOBEC3B expression in breast cancer reflects cellular proliferation, while a deletion polymorphism is associated with immune activation
APOBEC3B-Mediated Cytidine Deamination Is Required for Estrogen Receptor Action in Breast Cancer
APOBEC3G genotypes and proviral DNA hypermutations on HIV/AIDS disease progression in Thai and Cambodian children
APOBEC-Induced Cancer Mutations Are Uniquely Enriched in Early-Replicating, Gene-Dense, and Active Chromatin Regions
APOBECs and virus restriction
APOBEC3G expression is dysregulated in primary HIV-1 infection and polymorphic variants influence CD4+ T-cell counts and plasma viral load
APOBEC3G Generates Nonsense Mutations in Human T-Cell Leukemia Virus Type 1 Proviral Genomes In Vivo
APOBEC-1-mediated RNA editing
APOBEC-1 Complementation Factor (ACF) forms RNA-dependent multimers
APOBEC3 Proteins Expressed in Mammary Epithelial Cells Are Packaged into Retroviruses and Can Restrict Transmission of Milk-Borne Virions
APOB-516 T allele homozygous subjects are unresponsive to dietary changes in a three-month primary intervention study targeted to reduce fat intake
APOBEC3G Expression Correlates with T-Cell Infiltration and Improved Clinical Outcomes in High-grade Serous Ovarian Carcinoma
APOBEC3A damages the cellular genome during DNA replication
APOBEC3DE Inhibits LINE-1 Retrotransposition by Interacting with ORF1p and Influencing LINE Reverse Transcriptase Activity
APOBEC3G and APOBEC3F Act in Concert To Extinguish HIV-1 Replication
APOBEC3 proteins can copackage and comutate HIV-1 genomes
APOBEC3B high expression status is associated with aggressive phenotype in Japanese breast cancers
APOBEC4 Enhances the Replication of HIV-1
APOBEC3G Restricts HIV-1 to a Greater Extent than APOBEC3F and APOBEC3DE in Human Primary CD4(+) T Cells and Macrophages
APOBEC3B can impair genomic stability by inducing base substitutions in genomic DNA in human cells
APOBEC3A, APOBEC3B, and APOBEC3H Haplotype 2 Restrict Human T-Lymphotropic Virus Type 1
APOBEC3G expression and hypermutation are inversely associated with human immunodeficiency virus type 1 (HIV-1) burden in vivo
APOBEC3A Is a Specific Inhibitor of the Early Phases of HIV-1 Infection in Myeloid Cells
APOBEC3 proteins and genomic stability The high cost of a good defense
APOBEC3A can activate the DNA damage response and cause cell-cycle arrest
APOBEC3-mediated hypermutation of retroviral vectors produced from some retrovirus packaging cell lines
APOBEC1 and APOBEC3 Cytidine Deaminases as Restriction Factors for Hepadnaviral Genomes in Non-Humans In Vivo
APOBEC3 proteins mediate the clearance of foreign DNA from human cells
APOBEC3A catabolism of electroporated plasmid DNA in mouse muscle
APOBEC3A cytidine deaminase induces RNA editing in monocytes and macrophages
APOBEC3 inhibits DEAD-END function to regulate microRNA activity
APOBEC3F and APOBEC3G Inhibit HIV-1 DNA Integration by Different Mechanisms
APOBEC3G Contributes to HIV-1 Variation through Sublethal Mutagenesis
APOBEC proteins and intrinsic resistance to HIV-1 infection
APOBEC3G-Augmented Stem Cell Therapy to Modulate HIV Replication: A Computational Study
APOBEC3 Has Not Left an Evolutionary Footprint on the HIV-1 Genome
APOBEC3G: a double agent in defense
APOBEC deaminases-mutases with defensive roles for immunity
APOBEC3 proteins and reverse transcription
APOBEC3G Oligomerization Is Associated with the Inhibition of Both Alu and LINE-1 Retrotransposition
APOBEC3G: an intracellular centurion
APOBEC3G mRNA expression in exposed seronegative and early stage HIV infected individuals decreases with removal of exposure and with disease progression
APOBEC3G induces a hypermutation gradient: purifying selection at multiple steps during HIV-1 replication results in levels of G-to-A mutations that are high in DNA, intermediate in cellular viral RNA, and low in virion RNA
APOBEC3B and AID Have Similar Nuclear Import Mechanisms
APOBEC3G-independent reduction in virion infectivity during long-term HIV-1 replication in terminally differentiated macrophages
APOBEC3G cytidine deaminase association with coronavirus nucleocapsid protein
APOBEC3G is incorporated into virus-like particles by a direct interaction with HIV-1 Gag nucleocapsid protein
APOBEC3G incorporation into human immunodeficiency virus type 1 particles
APOBEC3G expression is restricted to epithelial cells of the proximal convoluted tubules and is not expressed in the glomeruli of Macaques
APOBEC3G expression is restricted to neurons in the brains of pigtailed macaques
APOBEC deaminases as cellular antiviral factors: A novel natural host defense mechanism
APOBEC3 cytidine deaminases: Distinct antiviral actions along the retroviral life cycle
APOBEC3G hypermutates genomic DNA and inhibits Ty1 retrotransposition in yeast
APOBEC3G Complexes Decrease Human Immunodeficiency Virus Type 1 Production
APOBEC3G Impairs the Multimerization of the HIV-1 Vif Protein in Living Cells
APOBEC3G is degraded by the proteasomal pathway in a vif-dependent manner without being polyubiquitylated