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Id-1 promotes migration and invasion of non-small cell lung cancer cells through activating NF-kappa B signaling pathway
Id-Like Reaction to BCG Therapy for Bladder Cancer
id-eCK Secure ID-Based Authenticated Key Exchange on Symmetric and Asymmetric Pairing
Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts
Id1 promotes lung cancer cell proliferation and tumor growth through Akt-related pathway
Id1 cooperates with oncogenic Ras to induce metastatic mammary carcinoma by subversion of the cellular senescence response
Id1 enhances human ovarian cancer endothelial progenitor cell angiogenesis via PI3K/Akt and NF-kappa B/MMP-2 signaling pathways
Id1 expression promotes peripheral CD4(+) T cell proliferation and survival upon TCR activation without co-stimulation
Id1 and NF-kappa B promote the generation of CD133(+) and BMI-1(+) keratinocytes and the growth of xenograft tumors in mice
Id1 Expression Promotes T Regulatory Cell Differentiation by Facilitating TCR Costimulation
Id1 and Id3 co-expression correlates with clinical outcome in stage III-N2 non-small cell lung cancer patients treated with definitive chemoradiotherapy
Id1 mRNA在卵巢癌患者内皮祖细胞中的表达及其临床意义
Id1 suppresses anti-tumour immune responses and promotes tumour progression by impairing myeloid cell maturation
Id1 overexpression is independent of repression and epigenetic silencing of tumor suppressor genes in melanoma
Id1 knockdown induces the apoptosis and inhibits the proliferation and invasion of ovarian cancer cells
Id1 enhances RING1b E3 ubiquitin ligase activity through the Mel-18/Bmi-1 polycomb group complex
Id1 and Id3 expression is associated with increasing grade of prostate cancer: Id3 preferentially regulates CDKN1B
Id1 Attenuates Notch Signaling and Impairs T-Cell Commitment by Elevating Deltex1 Expression
Id1 potentiates NF-kappa B activation upon T cell receptor signaling
Id1 Maintains Embryonic Stem Cell Self-Renewal by Up-Regulation of Nanog and Repression of Brachyury Expression
Id1 has a physiological role in regulating early B lymphopoiesis
Id1 immortalizes hematopoietic progenitors in vitro and promotes a myeloproliferative disease in vivo
Id1 Expression Level Determines the Differentiation of Human Dental Pulp Stem Cells
Id1 regulation of cellular senescence through transcriptional repression of p16/Ink4a
Id1 Deficiency Protects against Tumor Formation in Mice but Not in a Mouse Model of Colitis-Associated Colon Cancer
Id1 Represses Osteoclast-Dependent Transcription and Affects Bone Formation and Hematopoiesis
Id1 Is Down-Regulated by Hepatocyte Growth Factor via ERK-Dependent and ERK-Independent Signaling Pathways, Leading to Increased Expression of p16(INK4a) in Hepatoma Cells
Id1 Restrains p21 Expression to Control Endothelial Progenitor Cell Formation
Id1 restrains myeloid commitment, maintaining the self-renewal capacity of hematopoietic stem cells
Id1 induces the proliferation of cochlear sensory epithelial cells via the nuclear factor-kappa B/cyclin D1 pathway in vitro
Id1 transcription inhibitor-matrix metalloproteinase 9 axis enhances invasiveness of the breakpoint cluster region/abelson tyrosine kinase-transformed leukemia cells
Id1 gene expression and regulation in human thyroid tissue
Id1 gene transfer confers angiogenic property on fully differentiated endothelial cells and contributes to therapeutic angiogenesis
Id1, Id2 AND Id3 ARE INDUCED IN RAT MELANOTROPHS OF THE PITUITARY GLAND BY DOPAMINE SUPPRESSION UNDER CONTINUOUS STRESS
Id1, but not Id3, directs long-term repopulating hematopoietic stem-cell maintenance
Id1-induced inhibition of p53 facilitates endothelial cell migration and tube formation by regulating the expression of beta1-integrin
Id1-p53调控血管新生参与高切应力介导的易损斑块形成的机制
Id1/Id3 Knockdown Inhibits Metastatic Potential of Pancreatic Cancer
Id1/NR2B Receptor Pathway Regulates Rat Cochlear Sensory Epithelial Cell Survival after Radiation.
Id1: A novel therapeutic target for patients with atherosclerotic plaque rupture
Id1和E2-2参与血管内皮损伤修复机制的研究进展
Id1基因与妇科肿瘤相关性的研究
Id1在人肺癌细胞系A549肿瘤细胞球中的表达
Id1在肾细胞癌中的检测及其意义
Id1在肝细胞癌中的表达及其与癌细胞分化程度的相关性分析
Id2 expression delineates differential checkpoints in the genetic program of CD8 alpha(+) and CD103(+) dendritic cell lineages
Id2 promotes the invasive growth of MCF-7 and SKOV-3 cells by a novel mechanism independent of dimerization to basic helix-loop-helix factors
Id2 shRNA慢病毒载体的构建及其转染后对胶质瘤细胞增殖和凋亡的影响
Id2 Complexes with the SNAG Domain of Snai1 Inhibiting Snai1-Mediated Repression of Integrin beta 4
Id2 represses E2A-mediated activation of IL-10 expression in T cells
Id2 and Id3 maintain the regulatory T cell pool to suppress inflammatory disease
Id2 deletion attenuates Apc-deficient ileal tumor formation
Id2 Promotes Tumor Cell Migration and Invasion through Transcriptional Repression of Semaphorin 3F
Id2 deficiency promotes metastasis in a mouse model of ocular cancer
Id2 gene is a transcriptional target of the protein complex mutant p53/E2F1
Id2 regulates hyporesponsive invariant natural killer T cells
Id2 and E Proteins Orchestrate the Initiation and Maintenance of MLL-Rearranged Acute Myeloid Leukemia
Id2 reinforces T(H)1 differentiation and inhibits E2A to repress T-FH differentiation
Id2 Mediates Differentiation of Labyrinthine Placental Progenitor Cell Line, SM10